OCTOPAMINE-DEFICIENT MUTATION SUPPRESSES SHAKER PHENOTYPES IN DROSOPHILA P.K.Rivlin, J.L. Krans, K.D. Gawera, S.W. Wong and R.R.Hoy. Department of Neurobiology. - ppt download
Biomolecules | Free Full-Text | Genes Responsible for H2S Production and Metabolism Are Involved in Learning and Memory in Drosophila melanogaster
Shaw and Shal voltage‐gated potassium channels mediate circadian changes in Drosophila clock neuron excitability - Smith - 2019 - The Journal of Physiology - Wiley Online Library
Differential localization of voltage-gated potassium channels during Drosophila metamorphosis
Shal and Shaker Differential Contribution to the K+ Currents in the Drosophila Mushroom Body Neurons | Journal of Neuroscience
A Hyperkinetic Redox Sensor Drives Flies to Sleep: Trends in Neurosciences
Targeted Attenuation of Electrical Activity in Drosophila Using a Genetically Modified K+ Channel: Neuron
Shaker Gene - an overview | ScienceDirect Topics
Lithium-Responsive Seizure-Like Hyperexcitability Is Caused by a Mutation in the Drosophila Voltage-Gated Sodium Channel Gene paralytic | eNeuro